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Amfast (Atenolol)

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Amfast is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins). Amfast is used to treat angina (chest pain) and hypertension (high blood pressure).

Other names for this medication:
Adipin, Altoma, Amchek, Amlong, Amlotin, Amlowin, Amnest, Ampilife, Amsten, Angil, Atamo, Atark, Atedine, Atemos, Atenex, Atenolol, Atoli, Atwin, Betanif, Beten, Calock, Camcard, Cardipin, Cardpin, Celbeta, Cemalo, Cheerten, Cinamat, Depten, Hibesor, Hipres, Joglit, Lodip, Losar, Niftene, Noldepin, Nusar, Opime, Ortibid, Pertenol, Stressban, Tensigard, Tensimin, Tentoric, Ziblok

Similar Products:
Diovan, Prandin, Biaxin, Norvasc, Actos

Also known as: Atenolol


{name} (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

{name} is used to treat angina (chest pain) and hypertension (high blood pressure).

{name} is also used to lower the risk of death after a heart attack.


A nurse or other trained health professional will give you this medicine. This medicine is given through a needle placed in one of your veins .

Detailed Amfast dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amfast are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


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Age and sex distribution in urolithiasis with special reference to the nature of calculi. All lysosomotropic drugs tested triggered lysosomal activation demonstrated by increased lysosotracker red (LTR) and lysosensor green staining, increased cathepsin activity, and increased LAMP2 staining. Here we show that octaploid embryos can develop to the blastocyst stage in vitro, but all fetus impaired immediately after implantation. Soon after this breakthrough it became clear that there should exist additional risk alleles of other genes in order to fully explain the proportion of AD attributable to genetic factors. The PCPs measured using the four methods also differed significantly, suggesting that each method measures the pressure at a different site in the pulmonary circulation.

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